KETO

For Ketosis and Hypoglycemia
Solution For Oral Administration
Liver Supported

Category:

Description

KETO is metabolized in the rumen and liver and it affects metabolites, hormones, liver composition, feed intake, and milk production as well as preventing excessive fat mobilization, imbalances in carbohydrate and fat metabolism, and thereby reducing the risk of ketosis.
KETO decreases the molar ratio of acetate to propionate in rumen Volatile Fatty Acids (VFA) because part of KETO is metabolized to propionate in the rumen.
Oral administration of KETO increases insulin by 200-400% within 30 min after drenching.
KETO decreases plasma concentrations of non-esterified fatty acids (NEFA) and Beta-Hydroxybutyrate (BHB), especially in early lactating cows with relatively high levels of NEFA.
KETO also reduces the Triacylglycerol (TG) content of the liver and the concentrations of ketone bodies in milk and hence, has anti-ketogenic properties.

Each 1 mL Contains:

  • Propylene Glycol
  • Sorbitol
  • Dextrose
  • Excipients

Content indicate that KETO increases energy balance of cows in early lactation.
Together with the anti-ketogenic properties of KETO, this suggests that KETO may reduce the risk of subclinical and clinical ketosis.
Treatment of ketosis and hypoglycemia.
It stimulates sluggish liver parenchyma cells and restores liver functions.
It improves feed metabolism.
To enhance growth & weight gain.
To enhance liver metabolism & reduce ill effects of aflatoxins.

Calf, Sheep, & Goat:
20-30 mL per Animal by drinking water daily for 2 days.

Cattle:
100-150 mL per Animal by drinking water daily for 2 days.

Additional information

Administration

For oral administration.

Withdrawal Period

None.

Storage

Store in airtight containers, at a moderate temperature (15-25 °C) and protect from direct sunlight.

Packing

KETO is packed in 1 L plastic bottle.

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